Hypertension is considered to be the most important risk factor for stroke. Whether systolic or diastolic blood pressure is high, the risk of cerebrovascular disease is very high. Long-term high blood pressure load can cause systemic small artery spasm, vascular wall hyaline degeneration and fibrosis ( commonly known as cerebral vascular sclerosis ), lumen stenosis irreversible lesions. Small brain arteries can also undergo a series of changes from spasm to sclerosis. However, the cerebrovascular wall itself is weak and more fragile after hardening, and long-term hypertension leads to the formation of small vascular microaneurysms. In emotional excitement or tension, sudden fluctuations in pressure in the vascular cavity, microaneurysms or fragile vessel wall rupture bleeding. Hypertension promotes large and medium-sized atherosclerosis and small atherosclerosis. Small atherosclerosis is conducive to thrombosis. Intracranial and extracranial atherosclerotic plaque shedding can cause cerebral embolism. Brain tissue softening after infarction can damage the blood vessels around the lesion secondary to hemorrhage after infarction.
