Cerebral thrombosis usually occurs on the basis of atherosclerosis. For example, blood vessels change through atherosclerosis and form unstable plaques. The fibrous cap outside the unstable plaque is thinner and there is lipid. In the case of hemodynamic changes or excessive plaque, plaque rupture may occur. After rupture, platelets and fibrinogen may accumulate, initiate coagulation mechanism and fibrinogen. Platelets are entangled to form in situ thrombosis. If the patient has in situ thrombosis, the blood supply of the distal blood vessels will be reduced, and the patient may have ischemic symptoms, which is called cerebral thrombosis.
Cerebral thrombosis is one of the main causes of cerebral infarction. In addition to cerebral thrombosis, another cause of infarction is embolism, such as heart to blood vessel embolism or artery to artery embolism. For cerebral infarction caused by cerebral thrombosis, antiplatelet aggregation and statins are needed in the acute phase.
